Cardiac Workload Mastery: CO and Hemodynamics

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Match ideas to cardiac function terms

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Rosemary

United States

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Cardiac Workload Mastery: CO and HemodynamicsOnline version Match ideas to cardiac function terms by Rosemary 1 Cardiac Output Foundations 2 Meds: Targeted Adjustments 3 Preload: Volume Status 4 Afterload: Pressure Against Which The LV Works 5 Contractility: Strength of the Heartbeat 6 Interventions and Consequences Cardiac Output (CO) = Stroke Volume (SV) × Heart Rate (HR) Stroke Volume (SV) Heart Rate (HR) Normal CO range: 4–8 L/min If HR is too fast, filling time decreases and SV may drop Adjust Heart Rate (HR) Adjust Stroke Volume (SV) Modify Preload Modify Afterload Modify Contractility Preload = ventricular stretch at end of diastole Preload determines how full the ventricle is before contraction CVP normal range: 2–6 mmHg More fluid increases preload and stretch (↑ workload) Overstretch can weaken contraction (HF concern) Afterload = resistance LV must pump against High blood pressure increases afterload Lowering afterload helps reduce workload ACE inhibitors can decrease afterload Vasodilators help reduce afterload Contractility = strength of the ventricular squeeze Negative inotropes decrease contractility Beta blockers are negative inotropes Positive inotropes increase contractility Digoxin is a positive inotrope Too much preload raises workload Too little preload reduces stroke volume High afterload increases myocardial work Decreased preload can be achieved with diuretics Decreased afterload with vasodilators can improve CO

Cardiac Workload Mastery: CO and HemodynamicsOnline version

Match ideas to cardiac function terms

by Rosemary

Cardiac Output Foundations

Meds: Targeted Adjustments

Preload: Volume Status

Afterload: Pressure Against Which The LV Works

Contractility: Strength of the Heartbeat

Interventions and Consequences

Cardiac Output (CO) = Stroke Volume (SV) × Heart Rate (HR)

Stroke Volume (SV)

Heart Rate (HR)

Normal CO range: 4–8 L/min

If HR is too fast, filling time decreases and SV may drop

Adjust Heart Rate (HR)

Adjust Stroke Volume (SV)

Modify Preload

Modify Afterload

Modify Contractility

Preload = ventricular stretch at end of diastole

Preload determines how full the ventricle is before contraction

CVP normal range: 2–6 mmHg

More fluid increases preload and stretch (↑ workload)

Overstretch can weaken contraction (HF concern)

Afterload = resistance LV must pump against

High blood pressure increases afterload

Lowering afterload helps reduce workload

ACE inhibitors can decrease afterload

Vasodilators help reduce afterload

Contractility = strength of the ventricular squeeze

Negative inotropes decrease contractility

Beta blockers are negative inotropes

Positive inotropes increase contractility

Digoxin is a positive inotrope

Too much preload raises workload

Too little preload reduces stroke volume

High afterload increases myocardial work

Decreased preload can be achieved with diuretics

Decreased afterload with vasodilators can improve CO

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Cardiac Workload Mastery: CO and Hemodynamics

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About this activity

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Common Hacks I

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Muscular system

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SEQUENCE WORDS

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Glands and Hormones

Cardiac Workload Mastery: CO and HemodynamicsOnline version

by Rosemary

Cardiac Output Foundations

Meds: Targeted Adjustments

Preload: Volume Status

Afterload: Pressure Against Which The LV Works

Contractility: Strength of the Heartbeat

Interventions and Consequences

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