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The Biological Theory of OCD

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The Biological Theory of OCD

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Created by

RA Lomax
RA Lomax
New Zealand

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The Biological Theory of OCD
 

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The Biological Theory of OCDOnline version

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by RA Lomax
1

Obsessive - Compulsive Disorder ( OCD ) likely develops from a of genetic factors , neurotransmitter imbalances , and abnormal brain activity . play a key role , as OCD often runs in families . in genes such as COMT and SERT can increase risk . affects dopamine breakdown in the prefrontal cortex , which is important for thinking , - making , and self - control . Changes in levels can alter the activity of cortico - striato - thalamo - cortical ( ) circuits , which are in OCD . regulates recycling , and variations can affect serotonin , influencing mood , anxiety , and behaviours .

2

Neurotransmitter further contribute to OCD . Low reduces communication between neurons , which can lead to thoughts and compulsions , while dysregulated overstimulates habit and reward , reinforcing repetitive behaviours .

3

Abnormal structure and function also play a role . The orbitofrontal cortex ( ) is often , producing excessive worry and checking behaviours . The drives urges to actions and can amplify OFC activity , creating a cycle of anxiety and compulsion . Dysfunction in the basal ganglia , which control and movement , makes compulsive behaviours harder to .

4

OCD arises from the of genetic predispositions , neurotransmitter , and overactive brain . Altered dopamine and serotonin affect circuits , which decision - making , habit control , and regulation , producing the and compulsions of the disorder . Rather than a single cause , OCD results from multiple factors working together .

 
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