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Vasodilatadores: mecanismos y farmacocinética

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Vasodilatadores: mecanismos y farmacocinética

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Mecanismos, clasificación y PK

Created by

Maria Ximena Aldana G.
Maria Ximena Aldana G.
Bolivia

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Vasodilatadores: mecanismos y farmacocinética
 

Vasodilatadores: mecanismos y farmacocinéticaOnline version

Mecanismos, clasificación y PK

by Maria Ximena Aldana G.
1

Nitrates like nitroglycerin cause vasodilation by releasing NO which increases cGMP in vascular smooth muscle.

2

Minoxidil reduces potassium channel activity to cause smooth muscle contraction.

3

Hydralazine primarily produces arteriolar vasodilation with little effect on veins.

4

Sodium nitroprusside only dilates veins, not arteries.

5

Nitroglycerin is primarily used to dilate renal arteries to improve diuresis.

6

Calcium channel blockers (example: nifedipine, verapamil) cause vasodilation by inhibiting L-type Ca2+ channels in vascular smooth muscle.

7

Minoxidil opens ATP-sensitive potassium channels in smooth muscle causing hyperpolarization and vasodilation.

8

All vasodilators primarily work by decreasing heart rate; there is no effect on blood vessels.

9

Hydralazine acts by inhibiting ACE to reduce angiotensin II.

10

Sodium nitroprusside dilates both arterioles and veins, reducing afterload and preload.

 
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