What is Cerebral Vasospasm and What Do We Do?

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SAH vasospasm basics

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Brianna

United States

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Froggy Jumps What is Cerebral Vasospasm and What Do We Do?Online version SAH vasospasm basics by Brianna 1 When does cerebral vasospasm typically occur after a subarachnoid hemorrhage (SAH)? a Only after 21 days b Within 24 hours of SAH c Commonly days 3–14 post-SAH 2 What is the main consequence of vasospasm in the brain? a Cerebral hypoperfusion leading to delayed ischemia b Immediate brain hemorrhage c Increased intracranial pressure only 3 Which medication is used to prevent cerebral vasospasm after SAH? a Nimodipine (Nimotop) 60 mg q4h or 30 mg q2h b Furosemide 20 mg daily c Atenolol 50 mg daily 4 What BP effect is associated with Nimodipine? a No hemodynamic effect b Possible decrease in blood pressure c Significant increase in BP 5 What diagnostic tool helps detect vasospasm via velocity measures? a Transcranial Doppler (TCD) b MRI with contrast c EEG 6 A TCD finding indicative of vasospasm is a Lindegaard ratio greater than what value? a >1 b >3 c >5 7 What does the Lindegaard ratio compare? a Mean MCA velocity to ipsilateral extracranial ICA velocity b Atrial to cerebral venous flow c Intra-arterial pressure to venous pressure 8 If TCDs are high and the patient is symptomatic, what is the next step? a Initiate diuretic therapy b Refer to Interventional Radiology (IR) for intraarterial verapamil c Increase Nimodipine dose 9 What is the purpose of intraarterial verapamil in this context? a Reduce ICP surgically b Treat cerebral vasospasm via vasodilation c Anticoagulate cerebral arteries 10 Which statement correctly links timing, symptoms, and treatment for vasospasm? a Vasospasm only occurs before day 3; treat with BP meds b Nimodipine is avoided in SAH management c Vasospasm can occur up to day 21; treat with Nimodipine and IR-verapamil if symptomatic

Froggy Jumps

What is Cerebral Vasospasm and What Do We Do?Online version

SAH vasospasm basics

by Brianna

When does cerebral vasospasm typically occur after a subarachnoid hemorrhage (SAH)?

Only after 21 days

Within 24 hours of SAH

Commonly days 3–14 post-SAH

What is the main consequence of vasospasm in the brain?

Cerebral hypoperfusion leading to delayed ischemia

Immediate brain hemorrhage

Increased intracranial pressure only

Which medication is used to prevent cerebral vasospasm after SAH?

Nimodipine (Nimotop) 60 mg q4h or 30 mg q2h

Furosemide 20 mg daily

Atenolol 50 mg daily

What BP effect is associated with Nimodipine?

No hemodynamic effect

Possible decrease in blood pressure

Significant increase in BP

What diagnostic tool helps detect vasospasm via velocity measures?

Transcranial Doppler (TCD)

MRI with contrast

EEG

A TCD finding indicative of vasospasm is a Lindegaard ratio greater than what value?

What does the Lindegaard ratio compare?

Mean MCA velocity to ipsilateral extracranial ICA velocity

Atrial to cerebral venous flow

Intra-arterial pressure to venous pressure

If TCDs are high and the patient is symptomatic, what is the next step?

Initiate diuretic therapy

Refer to Interventional Radiology (IR) for intraarterial verapamil

Increase Nimodipine dose

What is the purpose of intraarterial verapamil in this context?

Reduce ICP surgically

Treat cerebral vasospasm via vasodilation

Anticoagulate cerebral arteries

Which statement correctly links timing, symptoms, and treatment for vasospasm?

Vasospasm only occurs before day 3; treat with BP meds

Nimodipine is avoided in SAH management

Vasospasm can occur up to day 21; treat with Nimodipine and IR-verapamil if symptomatic

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What is Cerebral Vasospasm and What Do We Do?

Froggy Jumps

About this activity

Created by

Download the paper version to play

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What is Cerebral Vasospasm and What Do We Do?Online version

by Brianna

When does cerebral vasospasm typically occur after a subarachnoid hemorrhage (SAH)?

What is the main consequence of vasospasm in the brain?

Which medication is used to prevent cerebral vasospasm after SAH?

What BP effect is associated with Nimodipine?

What diagnostic tool helps detect vasospasm via velocity measures?

A TCD finding indicative of vasospasm is a Lindegaard ratio greater than what value?

What does the Lindegaard ratio compare?

If TCDs are high and the patient is symptomatic, what is the next step?

What is the purpose of intraarterial verapamil in this context?

Which statement correctly links timing, symptoms, and treatment for vasospasm?

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