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Desgaste telomérico y hematopoyesis clonal

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Desgaste telomérico y hematopoyesis clonal

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Envejecimiento telomérico impulsa la CH en sangre.

Created by

Sayra
Sayra
Mexico

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Desgaste telomérico y hematopoyesis clonal
 

Desgaste telomérico y hematopoyesis clonalOnline version

Envejecimiento telomérico impulsa la CH en sangre.

by Sayra
1

En el UK Biobank, la CH provocada por DNMT3A, TET2 o JAK2 está relacionada con telómeros más cortos cuando el clon es grande.

2

TERTp-CH no muestra relación alguna con la longitud de los telómeros ni con la edad de aparición.

3

LOX y LOY no se asocian con cambios en la longitud de los telómeros ni con LTL-PRS.

4

Mutaciones en SF3B1, SRSF2 y U2AF1 son impulsores de CH y se asocian al envejecimiento.

5

La mayor parte de los casos de CH por PPM1D se deben a antecedentes de quimioterapia o radioterapia.

6

La CH inducida por SF3B1 y SRSF2 siempre presenta telómeros más cortos que los controles.

7

Las mutaciones en POT1 son responsables de CH de aparición tardía con telómeros perpetuamente largos.

8

En filogenias de HSPC, algunas colonias con SF3B1 y U2AF1 pueden mostrar telómeros relativamente largos.

9

Análisis de Mendeliano (MR) sugiere asociación causal entre menor longitud de telómeros y CH impulsada por PPM1D, SF3B1/SRSF2 y LOY.

10

El desgaste de los telómeros se convierte en un mecanismo de selección clonal en las HSC envejecidas.

 
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