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Ascitis: preguntas rápidas

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Preguntas sobre la fisiopatología de la ascitis

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Mexico

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Ascitis: preguntas rápidas
 

Ascitis: preguntas rápidasOnline version

Preguntas sobre la fisiopatología de la ascitis

by Mabel Ramírez
1

Hay hiperalbuminemia en estos pacientes.

2

La hipertensión portal aumenta la resistencia al flujo portal.

3

La retención renal de sodio y agua no participa en la ascitis.

4

El aumento de la presión hidrostática favorece la filtración de líquido hacia el espacio peritoneal.

5

El hígado cirrótico reduce la síntesis de albúmina, causando hipoproteinemia.

6

La ascitis se debe a la acumulación de líquido en cavidad peritoneal por desequilibrio entre formación y absorción.

7

La hipertensión portal no afecta la filtración renal.

8

La ascitis se produce por aumento de la presión osmótica en plasma.

9

La activación de los sistemas vasocativos aumenta la retención de sodio y agua (SRAA y ADH).

10

La vasodilatación esplácnica provoca aumento de volumen arterial efectivo sin cambios en la retención de sodio.

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