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ECG & Cardiac Rhythm Essentials

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Test your ECG and rhythm basics knowledge.

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ECG & Cardiac Rhythm Essentials
 

ECG & Cardiac Rhythm EssentialsOnline version

Test your ECG and rhythm basics knowledge.

by Maurice Kendricks
1

Which ECG interval reflects AV nodal conduction time (SA to AV)?

2

On standard ECG paper, one large box equals?

3

QT interval > 0.45 sec increases risk for?

4

Sinus bradycardia best describes?

5

Likely cause of newly symptomatic sinus bradycardia?

6

Unstable sinus bradycardia first-line medication/dose?

7

Sinus tachycardia in post-op patient with fever is most likely due to?

8

Priority action for stable narrow-complex SVT before meds?

9

Persistent SVT after vagal maneuvers: best sequence?

10

Premature atrial contractions (PACs) are typically?

11

Most important labs for frequent PVCs initially?

12

Assessment finding most expected with frequent PVCs?

13

Defining ECG feature of atrial fibrillation?

14

Highest priority risk in uncontrolled A-fib?

15

Which order requires bleeding precautions?

16

For A-fib >48 hours, elective cardioversion needs?

17

Atrial flutter key ECG pattern?

18

Aortic stenosis pathophysiology?

19

Classic triad of symptomatic severe AS?

20

Which finding is most consistent with aortic regurgitation?

21

Mitral stenosis hallmark auscultation?

22

Mitral regurgitation typical manifestation?

23

Tricuspid regurgitation assessment you'd expect?

24

First-line diagnostic to grade valvular disease severity?

25

Priority nursing management for symptomatic valvular HF exacerbation?

26

Typical presentation of acute pericarditis?

27

ECG change typical in acute pericarditis?

28

Pharmacologic mainstays for uncomplicated pericarditis?

29

Pericardial effusion ECG hallmark with large effusions?

30

Hemodynamically significant effusion priority intervention?

Feedback

PR reflects AV nodal conduction; QRS is ventricular, QT is repolarization, ST is isoelectric.

One large box is 0.20 s; small box is 0.04 s.

Prolonged QT raises risk of torsades and other malignant rhythms.

Bradycardia is a slow sinus rate; atrial/AV or ventricular origins are not correct here.

Negative chronotropes slow AV/SA conduction, causing bradycardia.

First-line is atropine; if ineffective, pacing or other agents considered.

Fever commonly drives sinus tachycardia; other options are less likely here.

Vagal maneuvers are first-line for stable SVT to terminate rhythm.

Adenosine is first-line acute termination for many SVTs after vagal attempts.

PACs are usually benign; consider rate control if frequent.

Electrolyte abnormalities, especially potassium and magnesium, predispose to PVCs.

PVCs can cause irregular beats; frequent PVCs may show compensatory patterns, not deficit.

AF has irregular rhythm and no discrete P waves.

Embolic stroke risk drives urgent anticoagulation decisions.

Direct oral anticoagulants require bleeding precautions.

To reduce embolic risk, anticoagulate or exclude atrial thrombus with TEE.

Atrial flutter shows sawtooth atrial activity.

AS is an outflow obstruction at the aortic valve.

SAD represents exertional symptoms of severe AS.

AR causes wide pulse pressure and bounding pulses.

OS followed by diastolic rumble is classic MS.

MR presents with holosystolic murmur to axilla and may have S3.

TR intensity increases with inspiration (Carvallo sign) and systemic signs present.

Echocardiography assesses structure and function of valves.

HF management focuses on symptoms and fluid balance.

Pain improves leaning forward; friction rub is common.

Diffuse ST elevation with PR depression is characteristic.

NSAIDs and colchicine are first-line; taper as symptoms improve.

Low voltage and beat-to-beat QRS changes (electrical alternans) seen with large effusions.

Significant effusion requires drainage and monitoring for tamponade signs.

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