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Fagocitosis y Mecanismos de Destrucción

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Fagocitosis y Mecanismos de Destrucción

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Conceptos clave de fagocitosis y opsoninas

Created by

Jose Luis
Jose Luis
Mexico

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Fagocitosis y Mecanismos de Destrucción
 

Fagocitosis y Mecanismos de DestrucciónOnline version

Conceptos clave de fagocitosis y opsoninas

by Jose Luis
1

1) ¿Qué células son principales fagocitos en inflamación (M1)?

2

2) ¿Qué son las opsoninas?

3

3) ¿Qué receptor reconoce la porción Fc de inmunoglobulinas para fagocitosis?

4

4) ¿Qué incluye la destrucción dependiente de oxígeno?

5

5) ¿Qué mecanismo describe la destrucción independiente de oxígeno?

6

6) ¿Qué tipo de receptores favorecen la fagocitosis sin opsonización?

7

7) ¿Cuáles son las opsoninas más importantes?

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8) ¿Qué citocinas inducen la activación M1 en macrófagos?

9

9) ¿Qué función principal tiene la fagocitosis por neutrófilos?

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10) ¿Diferencia entre oxidativo e independiente de oxígeno?

Explicación

Los neutrófilos y macrófagos M1 fagocitan y destruyen patógenos.

Las opsoninas facilitan la fijación de fagocitos a microbios.

Los Fc receptors permiten fagocitar opsonizados con IgG.

NADPH oxidasa genera ROS que matan microbios.

Se degradan patógenos mediante enzimas lisosomales sin depender de ROS.

PRR reconocen patrones moleculares y permiten fagocitosis sin opsoninas.

IgG y C3b facilitan la opsonización para fagocitosis.

IFN-γ y LPS promueven metabolismo inflamatorio M1.

Neutrófilos realizan phagocytosis rápida y muerte microbiana.

Oxidativo usa ROS; independiente usa enzimas lisosomales.

 
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