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Raíz del RAAS: Evaluación rápida

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Raíz del RAAS: Evaluación rápida

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Quiz sobre el sistema RAAS

Created by

Johan Angarita Alfonso
Johan Angarita Alfonso
Colombia

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Raíz del RAAS: Evaluación rápida
 

Raíz del RAAS: Evaluación rápidaOnline version

Quiz sobre el sistema RAAS

by Johan Angarita Alfonso
1

¿Qué enzima convierte angiotensinógeno en angiotensina I?

2

¿Dónde actúa principalmente la ACE para generar Angiotensina II?

3

¿Qué efecto principal de Angiotensina II en vasos sanguíneos?

4

¿Qué hormona aumenta la reabsorción de sodio en el túbulo distal?

5

¿Qué receptor media la acción vascular de Angiotensina II?

6

¿Qué libera renina en respuesta a baja perfusión renal?

7

¿Qué regula la liberación de Renina además de perfusión?

8

¿Qué órgano convierte Angiotensina I en Angiotensina II?

9

¿Qué resultado global promueve el RAAS si no hay regulación?

10

¿Qué efecto tienen los inhibidores de la ACE?

Explicación

La renina es la proteasa renal que inicia la vía, cortando angiotensinógeno en angiotensina I.

La ACE está muy expresada en endotelio pulmonar y convierte Ang I en Ang II.

Ang II produce vasoconstricción, aumentando la presión arterial.

Aldosterona incrementa la reabsorción de Na+ y excreción de K+.

Los efectos hemodinámicos de Ang II se asocian principalmente con el receptor AT1.

Las células yuxtaglomerulares secretan renina ante menor perfusión.

La macula densa en el ascendente del asa detecta NaCl y modula renina.

La ACE en endotelio pulmonar y vascular convierte Ang I en Ang II.

RAAS favorece presión arterial elevada y retención de Na+ y agua.

Inhiben ACE: bajan Ang II y pueden elevar bradicinina, favoreciendo vasodilatación.

 
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