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Hiperfunción adrenal: Cushing, Conn y CAH

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Hiperfunción adrenal: Cushing, Conn y CAH

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Quiz sobre síndromes adrenales

Created by

Jasble Ruiz
Jasble Ruiz
Colombia

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Hiperfunción adrenal: Cushing, Conn y CAH
 

Hiperfunción adrenal: Cushing, Conn y CAHOnline version

Quiz sobre síndromes adrenales

by Jasble Ruiz
1

¿Cuál es la causa principal de la síndorme de Cushing endógena?

2

En el síndrome de Conn, ¿qué hormona está elevada?

3

La hiperplasia suprarrenal congénita (CAH) suele deberse a defectos en

4

Un signo común de Cushing es

5

En el síndrome de Conn suele haber

6

La CAH puede presentar genitales ambiguos en 46,XX debido a

7

Tratamiento inicial recomendado para Cushing dependiente de la causa es

8

Una prueba diagnóstica para Cushing es

9

Una complicación típica del hiperaldosteronismo es

10

Diferencia clave entre Cushing y CAH es que Cushing

Explicación

El cortisol elevado por disfunción del eje HPA es clave en Cushing.

Conn se caracteriza por aldosterona alta, causando retención de sodio.

La deficiencia de 21-hidroxilasa es la causa más común de CAH.

La redistribución de grasa produce obesidad central en Cushing.

Aldosterona alta provoca pérdida de potasio (hipopotasemia).

La deficiencia enzimática eleva andrógenos, afectando genitales. aumenta virilización.

Eliminar la fuente de cortisol excesivo es clave.

Se evalúa la diurnalidad y la supresión de cortisol.

Aldosterona excesiva eleva presión y baja potasio.

Cushing centralmente es cortisol; CAH es enzimática con androgenización.

 
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