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Enfermedades suprarrenales y pheochromocitoma

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Enfermedades suprarrenales y pheochromocitoma

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Quiz sobre Addison, insuf. suprarrenal secundaria y feocromocitoma

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Leydi
Leydi
Colombia

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Enfermedades suprarrenales y pheochromocitoma
 

Enfermedades suprarrenales y pheochromocitomaOnline version

Quiz sobre Addison, insuf. suprarrenal secundaria y feocromocitoma

by Leydi
1

¿Qué caracteriza a la enfermedad de Addison?

2

¿Cuál es la causa más común de Addison en países desarrollados?

3

Qué síntoma es típico de Addison?

4

Diferencia clave entre Addison y insuficiencia suprarrenal secundaria?

5

Qué indica una prueba de estimulación con ACTH no eleva el cortisol en un paciente sospechoso?

6

Qué es un feocromocitoma?

7

Síntomas típicos de una crisis por feocromocitoma incluyen:

8

Tratamiento estándar de la Addison crónica?

9

Qué pruebas bioquímicas ayudan a confirmar un feocromocitoma?

10

Por qué es importante manejar el estrés en Addison?

Explicación

Es la insuficiencia primaria de la corteza suprarrenal con baja cortisol y, a menudo, hipopotasemia. La hiperpigmentación puede estar presente.

La autoinmunidad es la causa más frecuente en adultos; la TB era tradicional en otros contextos.

La hiperpigmentación es característica por exceso de ACTH en la insuficiencia primaria.

La insuf. secundaria suele mantener mineralocorticoides relativamente normales; la pigmentación suele ser menor o ausente.

Falla en la respuesta de cortisol ante ACTH sugiere fallo de la corteza renal (Addison).

Este tumor genera crisis hipertensivas por liberación de adrenalina y noradrenalina.

La tríada clásica es cefalea, palpitaciones y sudoración, a menudo con hipertensión.

Se usa hydrocortisona o cortisona y, si hay deficiencia mineralocorticoide, florinef (fludrocortisona).

Las metanefrinas son highly sensibles para confirmar feocromocitoma.

Durante infecciones o cirugía se requieren dosis glucocorticoides exógenas para evitar crisis.

 
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