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NBME Cardiology Quiz: Heart Essentials

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NBME Cardiology Quiz: Heart Essentials

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Cardiology NBME-style questions

Created by

Jazarah
Jazarah
Barbados

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NBME Cardiology Quiz: Heart Essentials
 

NBME Cardiology Quiz: Heart EssentialsOnline version

Cardiology NBME-style questions

by Jazarah
1

Which vessel supplies the majority of the interventricular septum via the posterior descending artery in most individuals?

2

Which heart structure is most associated with embryologic neural crest cells contributing to outflow tract formation?

3

What is the primary effect of ACE inhibitors in heart failure pharmacology?

4

Which congenital defect is characterized by a systolic murmur best heard at the left upper sternal border due to increased pulmonary flow?

5

Which ion channel mutation is commonly linked to long QT syndrome type 2?

6

Which defect involves an abnormal connection between the left and right atria due to incomplete septation?

7

What pharmacologic agent is preferred for rate control in atrial fibrillation with fast ventricular response in heart failure?

8

Which congenital heart disease is classically associated with a boot-shaped heart on radiographs?

9

In embryology, which structure forms the smooth part of the right ventricle and pulmonary trunk?

10

Which defect features a narrowed aorta distal to the left subclavian artery?

Feedback

In 70% of people, PDA arises from RCA; in others from LCx. PDA supplies inferior/septal region.

Neural crest cells populate the truncus arteriosus and conotruncal cushions forming the aorta and pulmonary trunk.

ACE inhibitors lower angiotensin II, dilate efferent arterioles, reduce afterload, and slow remodeling.

ASD causes fixed split S2 and systolic murmur from increased flow across pulmonary valve.

LQTS2 due to hERG channel dysfunction reducing IKr repolarizing current.

ASD results from incomplete fusion of septum primum and secundum.

Beta-blockers control rate; avoid in decompensated HF; alternatives include digoxin or non-dihydropyridine calcium blockers with caution.

TOF shows tetralogy features; boot-shaped due to right ventricular hypertrophy.

Bulbus cordis contributes to conus arteriosus becoming smooth outflow tract.

Coarctation typically near ductus arteriosus insertion; presents with differential pulses.

 
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