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Splenorenal ligament anchors spleen to stomach and left kidney.
Spleen resides in the LUQ beneath the diaphragm.
White pulp handles lymphatic/immune functions.
Red pulp contains splenic sinuses and cords for phagocytosis.
Spleen distends to store blood in venous sinuses.
Splenic artery is a branch of the celiac trunk.
Splenic vein joins SMV to form the MPV.
Wandering spleen is ectopic due to lax ligaments.
Accessory spleen is also called splenunculus.
Asplenia means spleen failure to develop.
Polysplenial pattern with multiple small spleens.
Asplenia can show situs anomalies including left-sided liver.
Retrorenal describes spleen behind kidney pole.
Pitting cleans RBCs by removing inclusions.
Culling removes irregular RBCs from circulation.
Hemoglobin breakdown forms biliverdin then bilirubin.
Splenic artery originates from the celiac trunk.
Long axis optimizes splenic visualization.
White pulp comprises about a quarter of spleen.
Red pulp handles phagocytosis and filtration.
Asplenia can be associated with right-sidedness.
Spleen length typically 8–12 cm.
Gastrosplenic ligament connects spleen to stomach.
White pulp is lymphatic tissue producing lymphocytes.
Spleen is part of the lymphatic immune system.
Accessory spleens are common incidental findings.
Splenic vein joins SMV to form the MPV.
Spleen tends to decrease in size with age.
Splenomegaly is enlargement of the spleen.
Spleen filters blood and recycles old RBCs.
Gastrosplenic ligament has two layers separating sacs.
Splenic agenesis is a type of heterotaxia.
The spleen sits in the LUQ between stomach and diaphragm.
Spleen contributes by producing lymphocytes.
Retrorenal spleen can resemble a mass near the kidney.
Long-axis views optimize splenic vessel visualization.
Spleen has white pulp (immune) and red pulp (phagocytic).
Spleen acts as a reservoir for blood when distended.
Cords of Billroth are part of red pulp.
Accessory spleens share echogenicity and can enlarge.
Spleen participates in iron storage as part of RES.
Liver panel is not specific to spleen evaluation.
Spleen macrophages phagocytose aged RBCs.
Bilirubin is formed during heme breakdown.
Spleen has elastic lobules enabling distention.
Spleen is a primary lymphoid/immune organ, liver is not primary in immunity.
Massive splenomegaly can cause hypersplenism.
Spleen resides in LUQ near stomach.
Thrombocytosis is increased platelets.
Splenic artery is tortuous and runs along pancreas.
Splenic vein joins SMV to form MPV.
Accessory spleen is usually asymptomatic.
Bilirubin is bilirubin pigment from heme breakdown.
Normal male Hct ~39-50%.
Normal female Hct ~35-45%.
Retrorenal spleen is posterior to the kidney pole.
White pulp produces lymphocytes.
Spleen stores iron as part of RES functions.
Size tends to decrease with age.
Splenorenal ligament connects spleen to stomach and kidney.
Pitting removes inclusions from RBCs.
White pulp is lymphoid tissue with lymphocyte production.
Heterotaxia can involve abnormal laterality including liver/spleen positions.
Gastrophrenic is not a primary spleen ligament.
Hilum is portal for vessels and lymphatics.
Spleen is usually mid-to-low echogenic.
Accessory spleens share echogenicity.
Spleen contributes to defense by lymphocyte production.
Malpighian corpuscles are lymphoid tissue.
Normal spleen is homogeneous with moderate echogenicity.
Spleen participates in iron storage and platelet turnover.
Wandering spleen can cause torsion with intermittent pain.
Spleen processes phagocytosis and heme recycling.
Spleen is peritoneal except at hilum.
Portal hypertension can cause splenomegaly.
Spleen distends to hold blood in venous sinuses.
MPV is formed by SMV and splenic vein.
Splenectomy often causes thrombocytosis due to reduced pooling.
Spleen sits inferomedially near stomach and left kidney.
RBC breakdown in spleen contributes bilirubin production.
Splenic artery courses along pancreas to spleen.
Parenchyma is divided into white and red pulp.
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